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Targeted therapies are like *smart bombs* in the fight against TNBC, aiming directly at specific vulnerabilities in cancer cells. Remember, TNBC doesn't have those classic hormone receptors or HER2, so traditional targeted therapies don't work. But researchers are relentlessly pursuing new targets and developing innovative drugs to hit them. One exciting area is PARP inhibitors, which have shown real promise in patients with BRCA1 or BRCA2 mutations. These mutations impair a cell's ability to repair damaged DNA. PARP inhibitors block a protein called PARP, which is also involved in DNA repair. By blocking PARP, these drugs prevent cancer cells with BRCA mutations from repairing their DNA, ultimately leading to their death. Think of it as disabling their repair crew, leaving them vulnerable. Several PARP inhibitors, such as olaparib (Lynparza) and talazoparib (Talzenna), are now approved for use in patients with metastatic TNBC and BRCA mutations. Clinical trials have demonstrated that these drugs can significantly improve progression-free survival in this patient population.
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