diabetes verbotene lebensmittel - The root cause of Friedreich's Ataxia lies in a genetic mutation. Specifically, it involves a gene called FXN, which provides instructions for making a protein called frataxin. **Frataxin is crucial for the proper functioning of mitochondria**, the powerhouses of our cells. This protein helps regulate iron levels within mitochondria, ensuring they produce energy efficiently. In individuals with FA, there's a significant reduction in the amount of frataxin produced. This deficiency leads to iron buildup within mitochondria, causing oxidative stress and damage to cells, particularly in the nervous system and heart. The mutation in the FXN gene is an expansion of a specific DNA sequence (GAA repeats). Normally, this sequence is repeated a certain number of times, but in people with FA, the number of repeats is much higher, disrupting the gene's ability to produce enough frataxin.
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